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Procelac (Omeprazole)
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Omeprazole

Procelac is a proton pump inhibitor that inhibits secretion of gastric acid by irreversibly blocking the enzyme system of hydrogen/potassium adenosine triphosphatase, the “proton pump” of the gastric parietal cell. The drug is used in conditions, where the inhibition of gastric acid secretion may be beneficial, including aspiration syndromes, dyspepsia, gastro-oesophageal reflux disease, peptic ulcer disease, and the Zollinger–Ellison syndrome. EsProcelac that is an isomer of Procelac is also used. The dose of Procelac may need to be reduced in patients with hepatic impairment.

Other names for this medication:
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Knowledge and use of memory strategies in amnestic mild cognitive impairment. Serotonergic (5-HT) mechanisms may be involved in impulse control (including antisocial behavior) across psychiatric syndromes. The conditions for an optimal ophthalmic coagulating system are derived from the absorption spectra of xanthophyll, melanin and hemoglobin.

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Seasonal variation in seminal characteristics of West African dwarf sheep in the humid tropics. In nine of 10 patients with syringomyelia, there was a substantial increase in substance P immunoreactivity in the first, second, third, and fifth laminae below the level of the lesion. Profound studies of iatrogenesis from a medical and legal viewpoint are an important task. Bioluminescent imaging (BLI) to improve and refine traditional murine models of tumor growth and metastasis.

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The treatment strategies applied to this population are presented in 3 time periods (1984-1990, 1991-1993, and 1994-1998). Cine magnetic resonance imaging and color Doppler flow mapping in infants and children with pulmonary artery bands. Efficacy and safety of tamsulosin as a medical expulsive therapy for stones in children.

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Electron correlation in the self-trapped hole and exciton in the NaCl crystal. We retrospectively reviewed 344 patients who underwent kidney transplantation from 1973 to 2007 compared them with 863 subjects with HIV/AIDS at the HIV-Netherlands/Australia/Thailand Research Collaboration (HIV-NAT) from 1997 to 2007. Both CCl4-induced hematotoxicity and nephrotoxicity were observed in both rats and mice, but those toxicities were manifested at higher exposure concentrations than hepatotoxicity. These mutations cause ligand-independent constitutive activation of the tyrosine kinase receptor by the formation of disulfide-bonded homodimers. This study shows that low-dose bupivacaine (with sufentanil), combined with a low-dose infusion of phenylephrine and moderate cohydration, gives the best hemodynamic stability during spinal anesthesia for cesarean delivery.

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